Notes on NEET PG

1. Heart size is Normal in :  TOF and TAPVC ( infracardiac form ) 2. Visual reflex formation or Macula mature by  5-6 months life 3.  Gardasil  — 16 , 18 , 6 , 11    Cervarix — 16 , 18 4. Renal tubular acidosis —  Normal Anion gap Hyperchloremic Metabolic Acidosis     Point to remember — its  NORMAL  in  Serum  but  POSITIVE  anion gap in  Urine 5. Ectopic ACTH Syndrome —  Hypokalemic Alkalosis 6. Anatomical Closure of Ductus arteriosus —  1-3 months  after birth. 1-3 months after birth. (ref: Langman’s embryology & Guyton) 7. Corneal donor — till  6 hour  after death 8. Nerve fibers — A and B are  Myelinated ; A to C – Diameter and Velocity  decrease 9. For Congenital hypothyroidism — TSH , best done  >48 hrs – 6 days  life 10. Child of HIV positive Mother — Cant use IgG for  1st 18 months  to diagnose as that comes from   ...

What is associated with: Orphan Annie cells? Papillary carcinoma of the ovary What is associated with: Russell bodies? Multiple myeloma What isassociated with: Reinke’s crystals? Leydig cell tumor What is associated with: Blue sclera? Osteogenesis imperfecta What isassociated with: Soap-bubble appearance on an x-ray? Giant cell tumorof the bone What is associated with: Pseudorosettes? Ewing’s sarcoma What isassociated with: Lucid interval? Epidural hematoma What isassociated with: Bloody tap on lumbar puncture? Subarachnoid hemorrhage What is associated with: Pseudopalisades? Glioblastoma multiforme What isassociated with: Charcot-Leyden crystals? Bronchial asthma (eosinophil membranes) What is associated with: Cafe au fait spot on the skin? Neurofibromatosis What is associated with: Streaky ovaries? Turner’s syndrome What isassociated with: Keratin pearls? Squamous cell carcinoma What is ass...

      ACUTE ALCOHOL INTOXICATION    NEET PG FREE EBOOKS NEET PG FREE NOTES           Consider acute alcohol intoxication in any patient presenting with: -coma -syncope -any inappropriate behavior -any neurological abnormality -trauma -traffic accident -hypothermia Inappropriate Behavior ; in coordination, confusion, slurred speech, aggressive, inappropriately sensitive behavior etc Neurological Abnormality; e.g. ataxia, nystagmus, divergent bulbae etc. Reliable history gives more helpful information in contrast to physical examination which is less sensitive and specific. MANAGEMENT                 Assess vital functions                              ...

1.When should a case of Non-Insulin dependent diabetes mellitus (NIDDM) with a history of diabetes for one year have an ophthalmic examination? A. As early as feasible B. After 5 years C. After 10 years D. Only after visual symptoms develop Correct answer : A. As early as feasible The person may have been diabetic for some time before the diagnosis of NIDDM. Retinopathy may have already occured. Hence, ophthalmological testing should be done as early as possible after diagnosis of NIDDM. If there are no pathologic changes, he should come for review every 6 months / 1 year. But in case of IDDM (Insulin dependant diabetes mellitus), the patients are diagnosed early in life. There is much less chance of any ophthalmologic disease at the time of diagnosis. Hence ophthalmologic examination is recommended 5 years after the diagnosis of IDDM. 2 Which one of the following would be expected in pyruvate kinase deficiency? (A) Increased levels of lactate in the R.B.C (B) Hemolytic ane...

1.Complications of stellate ganglion block are all except? A. Mediastinitis B. Cardiac arrest C. Pneumothorax D. Mueller’s syndrome Correct answer : D. Mueller’s syndrome Mueller’s syndrome is a sign of successful stellate ganglion block in which there is warmth of the face and injection of the tympanic membrane. 2.Which nutritional deficiency is associated with hereditary ataxia? A. Vitamin B12 B. Vitamin B1 C. Vitamin E D. Vitamin C Correct answer: C. Vitamin E Explanation: There are two types of hereditary ataxia associated with abnormalities of Vitamin E. Abetalipoproteinemia – Impairment of formation and secretion of VLDL in liver resulting in deficiency of delivery of Vitamin E to tissues including central and peripheral nervous system. Ataxia with Vitamin E deficiency (AVED) – Impaired binding of Vitamin E to VLDL, resulting in deficiency of Vitamin E in peripheral tissues. Reference: Harrison’s Principles of Internal Medicine, 19th edition, p2630 ...